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L664R

Category 3/4 — Most DruggableConflictingLumenal · predictedσ-1 candidateEditorial
LeucineArginine at position 664 · C-terminal lumenal domain (653-869) · WFS1 (Wolframin)

Leucine → Arginine at position 664 in wolframin's C-terminal lumenal domain. ClinVar Conflicting classifications including Wolfram syndrome 1. AlphaMissense 0.980, DynaMut2 ΔΔG -0.75 kcal/mol (destabilising). Charge introduction into a hydrophobic position.

Interactive 3D Structure

Wild-type reference
Wild-type L664 — hydrogen bond to S662
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DynaMut2 mutant · L664R
Mutant R664 — van der waals to S662 lost (6 contacts lost)
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Bond changes · DynaMut2 interaction analysis

6 lost1 gained4 preserved
Interaction typeWild-type partnerMutant partnerStatus
Hydrogen bondS662S662Preserved
Hydrogen bondW700W700Preserved
Polar contactS662S662Preserved
Polar contactW700W700Preserved
Van der WaalsS662Lost
Van der WaalsW700Gained
HydrophobicQ668Lost
HydrophobicY669Lost
HydrophobicL693Lost
HydrophobicV698Lost
HydrophobicL829Lost

Lost / gained / preserved interatomic contacts at the variant residue, from the DynaMut2 (Arpeggio) interaction analysis of the wild-type and energy-minimized mutant structures.

Computational Predictions

DynaMut2 ΔΔG
-0.75kcal/mol
Destabilising — mild
AlphaMissense
0.980
LPath
AlphaFold pLDDT
85
model confidence
Schema
Cat 3/4
Category 3/4 — Most Druggable
ΔΔG confidence: Standard confidence · Lumenal

This variant is in a water-facing region, which is the regime the stability predictor was built and benchmarked for. Ordinary predictor error still applies.

Clinical Evidence

ClinVar classificationConflicting classifications of pathogenicity
Review statuscriteria provided, conflicting classifications
Associated conditionsWolfram syndrome 1
InheritanceWolfram syndrome 1 documented.
Population frequency (gnomAD v4)Ultra-rare · AF 0.000068%
cDNA changec.1991T>G
ClinVar accessionVCV001705303
Last evaluated2022/09/23 00:00

Observed at very low frequency in gnomAD.

Classified for1★ unverified submission

ClinVar classifies this variant as Conflicting classifications of pathogenicity for Wolfram syndrome (classic) (autosomal recessive, OMIM 222300). Classic Wolfram syndrome is recessive: a single copy does not produce it, and this classification says nothing about a carrier's own risk.

  • Wolfram syndrome (classic)autosomal recessive · OMIM 222300submitted as: Wolfram syndrome 1
  • Submitters conflict on this classification (1★). Treat the conditions above as submitted, not as documented phenotypes.
  • Phenotype scope is interpretation of submitted records. The computed values on this card (AlphaMissense, DynaMut2 ΔΔG, pLDDT) are measurements of the model, not of a patient.
  • Presentation cannot be generalised from neighbouring variants: one nucleotide over can present very differently. Structural similarity supports a mechanistic hypothesis, never a phenotype claim.

Review status: 1★ conflicting classifications. ClinVar "conditions" is the union of all submissions; 0–1★ entries are submitted conditions, not documented phenotypes.

Population frequency
Global (all ancestries)
AF 0.000068% · 1 / 1,461,158 alleles
Homozygotes
0

Every ancestry group with observed alleles falls below the reporting threshold (min 2 alleles, min 500 sampled) — too sparse to name a highest-frequency population.

No homozygotes reported in gnomAD v4. For a recessive allele this is expected and is not, on its own, evidence either way.

Ancestry groupAllele freq.AC / ANHom.Carrier est.
European (non-Finnish) · under-sampled0.000090%1 / 1,111,9960

Source: gnomAD r4, retrieved 2026-08-21. Values are allele frequencies. The carrier estimate is derived from the allele frequency (Hardy-Weinberg, ~2·AF) and is an approximation, not a published figure — where a published carrier frequency exists it outranks this column and is cited on the card.

Structural Context

Position 664 sits in wolframin's C-terminal lumenal domain. The AlphaFold model places L664 within 5 Å of THR665 (2.4 Å), THR663 (2.5 Å), GLN668 (3.7 Å — same Q668 as Y669 and L672P environment), SER662 (4.4 Å), and VAL698 (4.9 Å — long-range).

Replacing L664 with arginine introduces a large positive charge into a polar-leaning local environment with H-bonding partners (T665, T663, Q668, S662). The new R664 guanidinium can H-bond with these partners but pulls the local geometry into a new configuration. The Y669-C673-L672-Q668 microregion (densely populated by Atlas variants) is affected.

The |ΔΔG| of 0.75 reflects fold cost. AlphaMissense's 0.980 + Wolfram syndrome 1 confirm severe functional consequence.

Amino-acid chemistry
Leucine (L) → Arginine (R) — branched aliphatic hydrophobic replaced by large positively-charged guanidinium-bearing residue. Charge introduction into a non-polar environment.
Position in the protein
C-terminal lumenal domain · position 664 in the ER lumen (pLDDT 85).

Druggability Assessment

Category 3/4 — Most Druggable. |ΔΔG| = 0.75 — fold survives. AlphaMissense 0.980 + Wolfram 1 confirm severe functional consequence.

Mechanism is charge introduction into the polar 663-665 microregion that abuts the dense Y669-C673 cluster. Therapeutic strategy: site-directed at the polar microregion adjacent to the Y669 cluster.

Why this matters

L664R sits at the edge of the dense Y669-C673-L672 cluster — close enough that drug discovery in the cluster region likely engages L664 as well.
Therapeutic Strategy Handoff · prediction

Feed this card to Wolfram Intelligence

Download the L664R PDF below and upload it to Wolfram Intelligence to generate therapeutic-strategy proposals — guanidinium mimetics, sigma-1 agonist docking, NAC thiol-capping. NAC is already on the bench-testing list.

Download L664R PDF card ↓Strategies are AI-generated predictions, not validated therapeutics.

UniProt Domain Annotation

Chain1890 · Wolframin
Topological domain653869 · Lumenal