RareResearch.AI
← Back to atlas

K290T

Category 4 — Stable Fold, Function DisruptedUncertain significanceCytoplasmic · predictedSource card
LysineThreonine at position 290 · N-terminal cytoplasmic (intrinsically disordered) · WFS1 (Wolframin)

Interactive 3D Structure

Wild-type reference
Wild-type K290 — hydrogen bond to L286
Fullscreen ↗
DynaMut2 mutant · K290T
Mutant T290 — hydrogen bond contact to L286 lost
Fullscreen ↗

Bond changes · DynaMut2 interaction analysis

0 lost2 gained3 preserved
Interaction typeWild-type partnerMutant partnerStatus
Hydrogen bondL286L286Preserved
Polar contactL286L286Preserved
Polar contactP292Gained
Van der WaalsL286L286Preserved
Van der WaalsP292Gained

Lost / gained / preserved interatomic contacts at the variant residue, from the DynaMut2 (Arpeggio) interaction analysis of the wild-type and energy-minimized mutant structures.

Computational Predictions

DynaMut2 ΔΔG
-0.15kcal/mol
Destabilising — mild
AlphaMissense
0.458
ambiguous
AlphaFold pLDDT
54
model confidence
Schema
Cat 4
Category 4 — Stable Fold, Function Disrupted

Clinical Evidence

ClinVar classificationUncertain significance
Review statuscriteria provided, single submitter
Associated conditions
Population frequency (gnomAD v4)Ultra-rare · AF 0.00020%
cDNA changec.869A>C
ClinVar accessionVCV003342991
Last evaluated2023/04/06 00:00

Observed at very low frequency in gnomAD.

Full Variant Card

WFS1 Wolframin — K290T Variant Card

Molecular Atlas Pilot · RareResearch.AI · Generated by wolfram-variant-card skill

Lysine → Threonine at position 290. N-terminal cytoplasmic (intrinsically disordered). ClinVar Uncertain significance, AlphaMissense 0.458, DynaMut2 ΔΔG -0.15 kcal/mol (destabilising).


Identity

FieldValue
VariantK290T (p.Lysine290Threonine)
DNA changec.869A>C
Gene · ProteinWFS1 · Wolframin (890 aa)
UniProtO76024 · WFS1_HUMAN
ClinVar accessionVCV003342991
Amino acid changeLysine (K) → Threonine (T)

Structural Context

FieldValue
AlphaFold modelAF-O76024-F1, v6
pLDDT at residue 29053.69 — confident
DomainN-terminal cytoplasmic (intrinsically disordered)
Position contextN-terminal cytoplasmic (intrinsically disordered)
IDR flagNo — pLDDT above 50 threshold

UniProt features at this position:

(none catalogued)

Position 290 sits in N-terminal cytoplasmic (intrinsically disordered). The wild-type residue is positively charged (lysine — primary amine); the mutant is small polar (threonine — hydroxyl). The chemistry shift implies altered local packing, hydrogen-bonding, and/or electrostatics at this site.


Computational Predictions

AlphaMissense

FieldValue
am_pathogenicity0.4582
am_classambiguous
InterpretationLikely benign (threshold 0.564)

DynaMut2

FieldValue
ΔΔG (kcal/mol)-0.15 (Destabilising)
Job ID178094709785
Result URLhttps://biosig.lab.uq.edu.au/dynamut2/results_prediction/178094709785

Clinical Evidence

FieldValue
ClassificationUncertain significance
Review statuscriteria provided, single submitter
Last evaluated2023/04/06 00:00
InheritanceInheritance pattern not specified in ClinVar entry; WFS1 has both AD and AR presentations.
WFS1 variant landscapeK290T is 1 of ~326 pathogenic-spectrum variants in WFS1 (out of 2,243 catalogued in ClinVar)

(no conditions catalogued)


Research Path Decision Tree

ΔΔG < 2  + binding site affected   →  CATEGORY 3 — docking experiments
ΔΔG 2–4                            →  CATEGORY 2 — pharmacological chaperones
ΔΔG > 4                            →  CATEGORY 1 — gene therapy
pLDDT < 50                         →  CATEGORY 5 — IDR, experimental only
Stable fold + functional site hit  →  CATEGORY 4 — site-specific docking

Final Schema Categorization

Category 4 — Stable Fold, Function Disrupted

<strong>Category 4 — Stable Fold, Function Disrupted</strong><br/><br/>|ΔΔG|=0.15 negligible. Likely site-specific functional disruption — docking strategy.

Why this card matters. Wolframin's fold survives this substitution (|ΔΔG|=0.15 kcal/mol). The pathogenic signal is real — AlphaMissense places it at 0.458. Protein still folds, but a specific local site is broken. Pharmacological chaperones and small-molecule binders are the rational therapeutic vector.


Files in this folder

  • AF-O76024-F1-model_v6.pdb — AlphaFold structure
  • K290T_molstar_viewer.html — interactive 3D viewer (auto-highlights position 290 with ball-and-stick + neighbors within 5Å)
  • K290T_variant_card.md — this card (source of truth)
  • K290T_variant_card.html — styled printable card
  • K290T_dynamut2_summary.html — clean offline DynaMut2 result card
  • dynamut2_result.json — structured result data
  • dynamut2_result_page.html — local snapshot of the Biosig result page (asset URLs absolutized)
  • K290T_wildtype_interactions.pse / K290T_mutant_interactions.pse — PyMOL sessions

Generated by wolfram-variant-card skill · RareResearch.AI Molecular Atlas Every assumption documented. Every score sourced.

Therapeutic Strategy Handoff · prediction

Feed this card to Wolfram Intelligence

Download the K290T PDF below and upload it to Wolfram Intelligence to generate therapeutic-strategy proposals — guanidinium mimetics, sigma-1 agonist docking, NAC thiol-capping. NAC is already on the bench-testing list.

Download K290T PDF card ↓Strategies are AI-generated predictions, not validated therapeutics.