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S411F

Category 3/4 — Most DruggableUncertain significanceTransmembrane · predictedSource card
SerinePhenylalanine at position 411 · Transmembrane helix 4 · WFS1 (Wolframin)

Interactive 3D Structure

Wild-type reference
Wild-type S411 — hydrogen bond to F408
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DynaMut2 mutant · S411F
Mutant F411 — hydrogen bond to L637 lost (4 contacts lost)
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Bond changes · DynaMut2 interaction analysis

4 lost13 gained12 preserved
Interaction typeWild-type partnerMutant partnerStatus
Hydrogen bondS353S353Preserved
Hydrogen bondS356Lost
Hydrogen bondH407H407Preserved
Hydrogen bondF408F408Preserved
Hydrogen bondF414F414Preserved
Hydrogen bondV415V415Preserved
Hydrogen bondL637Lost
Polar contactS353Gained
Polar contactS356S356Preserved
Polar contactC360Gained
Polar contactH407H407Preserved
Polar contactF408F408Preserved
Polar contactL409Gained
Polar contactF413F413Preserved
Polar contactF414F414Preserved
Polar contactV415V415Preserved
CarbonylF414Gained
Van der WaalsS356S356Preserved
Van der WaalsM357Gained
Van der WaalsF408Gained
Van der WaalsL409Gained
Van der WaalsF413Lost
Van der WaalsF414Lost
HydrophobicM357Gained
HydrophobicC360Gained
HydrophobicH407Gained
HydrophobicF408Gained
HydrophobicV633Gained
HydrophobicL637Gained

Lost / gained / preserved interatomic contacts at the variant residue, from the DynaMut2 (Arpeggio) interaction analysis of the wild-type and energy-minimized mutant structures.

Computational Predictions

DynaMut2 ΔΔG
-1.08kcal/mol
Destabilising — moderate
AlphaMissense
0.982
likely pathogenic
AlphaFold pLDDT
94
model confidence
Schema
Cat 3/4
Category 3/4 — Most Druggable

Clinical Evidence

ClinVar classificationUncertain significance
Review statuscriteria provided, multiple submitters, no conflicts
Associated conditionsWolfram-like syndrome; Cataract 41; Autosomal dominant nonsyndromic hearing loss 6; Type 2 diabetes mellitus; Wolfram syndrome 1
Population frequency (gnomAD v4)Ultra-rare · AF 0.00089%
cDNA changec.1232C>T
ClinVar accessionVCV001438079
Last evaluated2025/08/11 00:00

Observed at very low frequency in gnomAD.

Full Variant Card

WFS1 Wolframin — S411F Variant Card

Molecular Atlas Pilot · RareResearch.AI · Generated by wolfram-variant-card skill

Serine → Phenylalanine at position 411. Transmembrane helix 4. ClinVar Uncertain significance, AlphaMissense 0.982, DynaMut2 ΔΔG -1.08 kcal/mol (destabilising).


Identity

FieldValue
VariantS411F (p.Serine411Phenylalanine)
DNA changec.1232C>T
Gene · ProteinWFS1 · Wolframin (890 aa)
UniProtO76024 · WFS1_HUMAN
ClinVar accessionVCV001438079
Amino acid changeSerine (S) → Phenylalanine (F)

Structural Context

FieldValue
AlphaFold modelAF-O76024-F1, v6
pLDDT at residue 41194.12 — well-folded
DomainTransmembrane helix 4
Position contextInside Transmembrane helix 4 · position 411 is bilayer-embedded
IDR flagNo — pLDDT above 50 threshold

UniProt features at this position:

(none catalogued)

Position 411 sits in a transmembrane helix (Transmembrane helix 4). Wolframin has eleven such helices anchoring it in the ER membrane; substitutions inside the bilayer-embedded segments can disrupt helix packing, lipid contacts, and the overall ER topology of the protein. The wild-type residue is small polar (serine — hydroxyl); the mutant is large aromatic hydrophobic (phenylalanine). The chemistry shift implies altered local packing, hydrogen-bonding, and/or electrostatics at this site.


Computational Predictions

AlphaMissense

FieldValue
am_pathogenicity0.9820
am_classlikely pathogenic
InterpretationLikely pathogenic (threshold 0.564)

DynaMut2

FieldValue
ΔΔG (kcal/mol)-1.08 (Destabilising)
Job ID178092092352
Result URLhttps://biosig.lab.uq.edu.au/dynamut2/results_prediction/178092092352

Clinical Evidence

FieldValue
ClassificationUncertain significance
Review statuscriteria provided, multiple submitters, no conflicts
Last evaluated2025/08/11 00:00
InheritanceAutosomal dominant pattern indicated by associated DFNA6/14/38 (WFS1 hearing loss 6).
WFS1 variant landscapeS411F is 1 of ~326 pathogenic-spectrum variants in WFS1 (out of 2,243 catalogued in ClinVar)
  • Wolfram-like syndrome
  • Cataract 41
  • Autosomal dominant nonsyndromic hearing loss 6
  • Type 2 diabetes mellitus
  • Wolfram syndrome 1

Research Path Decision Tree

ΔΔG < 2  + binding site affected   →  CATEGORY 3 — docking experiments
ΔΔG 2–4                            →  CATEGORY 2 — pharmacological chaperones
ΔΔG > 4                            →  CATEGORY 1 — gene therapy
pLDDT < 50                         →  CATEGORY 5 — IDR, experimental only
Stable fold + functional site hit  →  CATEGORY 4 — site-specific docking

Final Schema Categorization

Category 3/4 — Most Druggable

<strong>Category 3/4 — Most Druggable</strong><br/><br/>|ΔΔG|=1.08 < 2 kcal/mol (fold intact) + AlphaMissense 0.982 confirms functional impact. Specific local contacts disrupted — priority for docking and pharmacological chaperone screening.

Why this card matters. Wolframin's fold survives this substitution (|ΔΔG|=1.08 kcal/mol). The pathogenic signal is real — AlphaMissense places it at 0.982. Protein still folds, but a specific local site is broken. Pharmacological chaperones and small-molecule binders are the rational therapeutic vector.


Files in this folder

  • AF-O76024-F1-model_v6.pdb — AlphaFold structure
  • S411F_molstar_viewer.html — interactive 3D viewer (auto-highlights position 411 with ball-and-stick + neighbors within 5Å)
  • S411F_variant_card.md — this card (source of truth)
  • S411F_variant_card.html — styled printable card
  • S411F_dynamut2_summary.html — clean offline DynaMut2 result card
  • dynamut2_result.json — structured result data
  • dynamut2_result_page.html — local snapshot of the Biosig result page (asset URLs absolutized)
  • S411F_wildtype_interactions.pse / S411F_mutant_interactions.pse — PyMOL sessions

Generated by wolfram-variant-card skill · RareResearch.AI Molecular Atlas Every assumption documented. Every score sourced.

Therapeutic Strategy Handoff · prediction

Feed this card to Wolfram Intelligence

Download the S411F PDF below and upload it to Wolfram Intelligence to generate therapeutic-strategy proposals — guanidinium mimetics, sigma-1 agonist docking, NAC thiol-capping. NAC is already on the bench-testing list.

Download S411F PDF card ↓Strategies are AI-generated predictions, not validated therapeutics.